My Posts

GSK3b explained
GSK3b BDNF Depression Anxiety UPR REDD1 mTOR CHOP IGF-1 EGF WNT PKA CaMK2 PP2A PP2B
Suspected role of Chromium in disinhibition
Disinhibition Binge Eating Disorder Chromium
Dopamine signalling inhibits Akt, leading to activation of GSK3b
Dopamine GSK3b Akt Lithium
GSK3b the pacifier
GSK3b HIF1 NRF2
Astrocyte metabolic coupling in Tourette syndrome pathogenesis
Astrocytes Tourette syndrome
Hypoxia increases ROS production and activates Nrf2
Hypoxia Nrf2 Apnea Oxidative stress
Hypoxia and GSK3b
hypoxia GSK3b HIF-1 OCD Nrf2
Creatine activates AMPK and induces PGC1a
Creatine PGC1a AMPK
Mitochondrial oxidative stress: blood-brain barrier disruption
Oxidative stress
Hypoxia lowers selenium metabolism
Selenium SCLY Hypoxia
Fat processing depends on PGC-1a
PGC1a Fasting Fat oxidation NAD Methylation potential
Methylation potential and perception of pain
Methylation potential Pain TRPA1
H2S clearance and reversal of ETC, role of CQ10
H2S Prepulse inhibition CQ10 Startle
AMPK and fear extinction
AMPK Fear extinction
Redox homeostasis, DNA damage and OCD
OCD Oxidative Stress
Retinoic acid and NAD
NAD Retinoic acid
Distinction of Phosphatidylcholine Synthesis between CDP-Choline and PEMT pathways
Phospholipids CDP-choline PEMT
High level of Endocannabinoids in CSF in TS
Tourette Syndrome ECS
Elevated DHA and DHA/AA ratio in ASD
DHA Phospholipids
Nrf2 might be inhibiting heme synthesis
Heme Iron SFN ALAD
Blocking sequential subcircuits in their hyperglutamatergic CSTC loop halts tics
Tourette Syndrome CSTC
Low methylation potential leads to hypermethylation of genes
FADS2 Methylation potential Phospholipids hypomethylation hypermethylation
Low GSH may not allow the resolution of the inflammation
GSH Inflammation Resolvins PGE2 PGD2 Prostaglandin DHA EPA Lipoxin
Overview how inflammation is initiated and resolved
Inflammation Resoleomics EPA DHA PGE2 PGD2 resolvin E1 prostanoid D1 VEGF LOX-5 COX-2
DGLA is able to abolish tardive dyskinesia
DGLA GLA Linoleic acid Tardive dyskinesia OCD phospholipids Tourette Syndrome Heme Iron
Arachidonic acid and DHA are probably the true Essential Fatty Acids
Phospholipids ARA DHA EFA
Cases of Tourette’s Syndrome and Aripiprazole
Tourette Syndrome Aripiprazole
Arachidonic acid synthesis requires heme
Arachidonic acid Iron Heme ECS
Maternal PUFA imbalance and anxiety in offsprings
PUFA phospholipids anxiety
Canadian Guidelines for the Evidence Based treatment of Tourette Syndrome
Tourette Syndrome Guidelines
Altered phospholipid and creatine in OCD
OCD Phospholipids Creatine
Nrf2 activation depends on NAD status
Nrf2 NAD BACH1 SIRT6
Cannabidiol induces antioxidant pathways
CBD ECS NRF2 Oxidative stress BACH1
High folic acid intake dysregulates hepatic cholesterol
Folic acid Cholesterol Methylation
Tricyclic Antidepressant and ECS
ECS antidepressants
Paracetamol and ECS
Paracetamol Acetaminophen ECS FAAH Tics Thoughts
Quality of Life and a Surveillant Endocannabinoid System
ECS anxiety depression
Medicinal cannabis markedly reduced OCD
OCD Cannabis ECS Case report
Opposite effects of NAM and Melatonin
Melatonin Nicotinamide Methylation Oxidative stress
Neuropulse announcement
neuropulse Wearable
NNMT regulates methyl donor metabolism
NNMT Methylation BHMT AHCY MAT1A
MS and SAHH might be cuproenzymes?
SAHH Copper Methionine synthase
Copper and SAHH
Methylation SAHH Copper
A clinical trial launched for a wearable device for TS
Clinical trial Wearable
Iron deficiency in mothers is associated with tics disorder in children
Iron Tics Tourette syndrome
Uranium inhibits Nrf2 activity
Uranium Nrf2 CSE CBS H2S
Vitamin D and pediatric OCD
Case Report Vitamin D OCD
Oral Iron Administration
Iron Ferritin Anxiety Depression Concentration Insomnia
Trace elements and tics disorder
Zinc Copper Iron Tics Tourette syndrome
PANS and low ferritin
Ferritin Iron PANS